1-In normal cells an RTK receptor that binds to epidermal growth factor is present. When EGF is present in the extracellular space it binds to the receptor and initiates an RTK signal transduction cascade that ultimately tells the cell to undergo mitosis. Describe what would happen to cell division if a cell begins to express both the natural EGF receptor and a mutated version that is capable of binding to EGF. While both receptors can bind to EGF the mutated version cannot initiate receptor dimerization and autophosphorylation.
2-Why is it beneficial for a cell signaling pathway to be coupled to a MAP kinase cascade which essentially activates a number of MAP kinase proteins via phosphorylation?
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